Библиография (список литературы к главе 7)
1. Благова О.В., Недоступ А.В., Морозова Н.С. и др. Аритмогенная дисплазия правого желудочка: полиморфизм клинических проявлений // Кардиология. 2012. Т. 52, № 4. С. 85-94.
2. Благова О.В., Недоступ А.В., Седов В.П. и др. Некомпактный миокард как первичный феномен или следствие дисфункции миокарда: клинические маски синдрома // Кардиология. 2012. Т. 52, № 11. С. 17-26.
3. Митрофанова Л.Б., Моисеева О.М., Хащевская Д.А. и др. Некомпактный миокард левого желудочка. Клинико-морфологическое исследование // Арх. пат. 2016. Т. 78, № 2. С. 29-35.
4. Поляк М.Е., Мершина Е.А., Заклязьминская Е.В. Некомпактный миокард левого желудочка: симптом, синдром или вариант развития? // Рос. кардиол. журн. 2017. № 2. С. 106-113.
5. Руденская Г.Е., Тверская С.М., Чухрова А.Л. и др. Клинико-генеалогическое и молекулярно-генетическое исследование мышечной дистрофии Эмери-Дрейфуса // Журн. неврол. и психиатр. 2006. Т. 106, № 10. С. 58-85.
6. 2013 ESC guidelines on the management of stable coronary artery disease: the Task Force on the management of stable coronary artery disease of the European Society of Cardiology. Task Force Members // Eur. Heart J. 2013. Vol. 34, N 38. P. 2949-3003.
7. Accorsi F., Caruso G., Fiorilli R. et al. [Idiopathic dilatation of the atria: a syndrome? Description of a case of idiopathic biatrial dilatation, review of the literature and a proposal for a pathogenetic interpretation] // G. Ital. Cardiol. 1987. Vol. 17, N 10. P. 874-882.
8. Ackerman M.J., Priori S.G., Willems S. et al. HRS/EHRA expert consensus statement on the state of genetic testing for the channelopathies and cardiomyopathies: this document was developed as a partnership between the Heart Rhythm Society (HRS) and the European Heart Rhythm Association (EHRA) // Europace. 2011. Vol. 13, N 8. P. 1077-1109.
9. Akdis D., Saguner A.M., Shah K. et al. Sex hormones affect outcome in arrhythmogenic right ventricular cardiomyopathy/dysplasia: from a stem cell derived cardiomyocyte-based model to clinical biomarkers of disease outcome // Eur. Heart J. 2017. Vol. 38, N 19. P. 1498-1508.
10. Amzulescu M.S., Rousseau M.F., Ahn S.A. et al. Prognostic impact of hypertrabeculation and noncompaction phenotype in dilated cardiomyopathy: a CMR study // JACC Cardiovasc. Imaging. 2015. Vol. 8, N 8. P. 934-946.
11. Andreini D., Pontone G., Bogaert J. et al. Long-term prognostic value of cardiac magnetic resonance in left ventricle noncompaction: a prospective multicenter study // J. Am. Coll. Cardiol. 2016. Vol. 68, N 20. P. 2166-2181.
12. Andréoletti L., Lévêque N., Boulagnon C. et al. Viral causes of human myocarditis // Arch. Cardiovasc. Dis. 2009. Vol. 102, N 6-7. P. 559-568.
13. Angelini A., Thiene G., Boffa G.M. et al. Endomyocardial biopsy in right ventricular cardiomyopathy // Int. J. Cardiol. 1993. Vol. 40, N 3. P. 273-282.
14. Anguera Ferrando N., Pujadas Capmany R., Abardia Oliva X., Casasús Ramón A. [Familial association of arrhythmogenic dysplasia of the right ventricle and obstructive hypertrophic myocardiopathy] [Article in Spanish] // Med. Clin. (Barc.). 1993. Vol. 101, N 20. P. 798.
15. Anselme F., Moubarak G., Savouré A., Godin B. et al. Implantable cardioverter-defibrillators in lamin A/C mutation carriers with cardiac conduction disorders // Heart Rhythm. 2013. Vol. 10, N 10. P. 1492-1498.
16. António M., Costa C., Venâncio M. et al. Left ventricular noncompaction: analysis of a pediatric population [Article in English, Portuguese] // Rev. Port. Cardiol. 2011. Vol. 30, N 3. P. 295-311.
17. Aras D., Ozeke O., Cay S. et al. Arrhythmogenic noncompaction cardiomyopathy: is there an echocardiographic phenotypic overlap of two distinct cardiomyopathies? // J. Cardiovasc. Ultrasound. 2015. Vol. 23, N 3. P. 186-190.
18. Arbustini E., Disertori M., Narula J. Primary prevention of sudden arrhythmic death in dilated cardiomyopathy: current guidelines and risk stratification // JACC Heart Fail. 2017. Vol. 5, N 1. P. 39-43.
19. Arbustini E., Favalli V., Narula N. et al. Left ventricular noncompaction: a distinct genetic cardiomyopathy? // J. Am. Coll. Cardiol. 2016. Vol. 68, N 9. P. 949-966.
20. Arimura S., Okada T., Tezuka T. et al. Neuromuscular disease. DOK7 gene therapy benefits mouse models of diseases characterized by defects in the neuromuscular junction // Science. 2014. Vol. 345, N 6203. P. 1505-1508.