| Reduced or lacking peristalsis. "Splashing sound" in the distended loops, "falling drop sound". | In the beginning - intensification of peristalsis. Further on - reduced or lacking peristalsis, "splashing sound" in the distended loops, "falling drop sound". |
| | It does not change for a long time. | There is tachycardia from the onset of the disease. |
| | It does not change for a long time. | There is hypotension from the onset of the disease. |
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Roentgenological investigation | High diaphragm cupola, atelectases, exudate in the pleura are possible. Distended intestinal loops, "Kloiber's cups", prevalence of gas over fluid. | In the process development there is the same picture as in paralytic AIO. In contrastive investigation - there is steady retention of the contrast passage near the obstacle (before the obstacle - in antegrade passage, after the obstacle - in irrigoscopy). |
| Fluid in free abdominal cavity. Distended by the gas intestinal loops. Intralumen deposition of the fluid, widening of the lumen of the small intestine, thickening of its folds. | In the process development there is the same picture as in paralytic AIO. Presence of both enlarged and collapsed intestinal loops. Detection of a tumor, invaginate is possible. |
| Distended hyperemic loops, turbid exudate, fibrin layers. | In the process development there is the same picture as in paralytic AIO. Detection of hernial hili, adhesions, tumors is possible. Intestinal collapse lower the place of the obstacle. |
| | In high AIO there is intestinal collapse. In low AIO a tumor or compression from the outside are detected. |
Spastic obstruction in modern conditions occurs extremely rarely. The clinical picture manifests by a cramping pain. Vomiting is rarely observed. Abdominal distention is absent. In the anamnesis there are professional harms (contacts with heavy metals) or helminthiasis. | |
The basic signs of hemostatic obstruction with intestinal necrosis are acute pain, hypotension, bloody admixture in the stools ("raspberry jelly") |
With progression of any form of mechanical AIO the clinical picture more and more resembles paralytic AIO due to the secondary paralysis of the intestinal wall ("intestinal tiredness", hypokalemia). The clinical picture develops more rapidly and is more pronounced in high obstruction, especially in the jejunum. In all forms of AIO there is inevitable aggravation of endogenic intoxication, hypovolemia, pronounced impairments of water-electrolytic, protein, fat and carbohydrate balance, acido-basic status, as well as the development of peritonitis and polyorganic insufficiency. |