I. What are the most frequent clinical signs of malabsorption syndrome?
1. Steatorrhea.
2. Weight loss.
3. Diarrhea.
4. Bone pain.
5. Anemia.
6. Weakness.
7. Hypotension.
II. Impaired hepatic synthesis and/or excretion of conjugated bile salts in patients with acute or chronic liver disease may result in:
1. Impaired formation of micellar lipid.
2. Impaired absorption of vitamin D.
3. Impaired absorption of calcium.
4. Impaired absorption of iron.
5. Impaired absorption of water-soluble vitamins.
6. Impaired absorption of sugars.
7. Impaired absorption of aminoacids.
III. Reduced secretion of pancreatic enzymes leading to failure of intralumenal digestion may result from:
1. Resection of 70% of pancreas.
2. Chronic pancreatitis that is accompanied by loss of at least 90% of the secretory capacity of the pancreas.
3. Decreased secretion of secretin.
4. Decreased secretion of cholecystokinin.
5. Disaccharidase deficiency.
6. Severe impairment of amino acid absorption.
IV. Anemia in malabsorption syndrome results from the impaired absorption of the following substances:
1. I ron.
2. Vitamin B12.
3. Folate.
4. Vitamin K.
5. Free fatty acids.
6. Glycerol.
7. Calcium.
V. Resection or severe inflammation of the proximal intestine leads to predominant malabsorption of the following substances:
1. Calcium.
2. Water-soluble vitamins.
3. Monoglycerides.
4. Fatty acids.
5. Electrolytes.
6. Bile salts.
7. Water.
VI. Resection or severe inflammation of the distal intestine leads to predominant malabsorption of the following substances:
1. Calcium.
2. Water-soluble vitamins.
3. Monoglycerides.
4. Fatty acids.
5. Electrolytes.
6. Bile salts.
7. Water.